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In neuronal cell cultures, GHK-Cu demonstrates remarkable neuroprotective effects, reducing oxidative damage by 55% and enhancing cell survival under stress conditions by 40% compared to control administrations

Disorders of mitochondrial import, export and translation, including sideroblastic anemia with ataxia, EVEN-PLUS syndrome and mitochondrial complex I deficiency due to nucleotide-binding protein-like protein gene defect, have also been implicated in ISC biogenesis defects

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