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While DNA methylation does not have the flexibility required for the fine-tuning of gene regulation, its stability is perfect to ensure the permanent silencing of transposable elements

Although moderate levels of ROS act as signaling molecules in cellular processes such as differentiation, proliferation, and adaptive responses [153], excessive ROS accumulation induces lipid peroxidation, protein modification, and mitochondrial DNA damage, ultimately leading to cellular senescence, apoptosis, or dysfunction [154]

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Prevention of singlet oxygen-induced DNA damage by lipoate

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